例1,男性,35岁,机关干部,性格内向,患者于半年前无明显诱因出现不自主咬牙,发作时牙关紧咬不能控制,每次持续时间数秒不等。每日发作数次～数十次,近日发作频繁,发作期间神清,无舌咬破及大小便失禁。发作后自觉疲乏,工作效率下降,未经任何治疗而来诊。患者即往体健,无外伤史及脑炎史。查体内科及神经系统检查均无异常。家族史(-)。头颅CT检查正常,脑电地形图检查见左颞及两额6功率明显升高,经棘波分析证实为棘波。口服卡马西平治疗,一个月后症状明显减轻,三个月后症状消失,复查脑电地形图无明显异常,此后未再发作。 Example 1, male, 35-year-old cadres, introverted, introverted, patients with no obvious incentive to appear six months ago, involuntary teething, dental clenching can not control the attack, each duration ranging from a few seconds. Attacks several times a day ~ dozens of times, recent seizures frequent, clear during the attack, no tongue bite break and incontinence. After the onset of conscious fatigue, decreased work efficiency, without any treatment and consultation. Patients who go to health, no history of traumatic brain injury and encephalitis. Physical examination and neurological examination were normal. Family history (-). Head CT examination was normal, EEG topography showed left temporal and two amount of power was significantly increased, confirmed by spike analysis as spikes. Oral carbamazepine treatment, a month after the symptoms were significantly reduced, symptoms disappeared after three months, review EEG topography no obvious abnormalities, then no further attacks.