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去甲肾上腺素（ＮＥ）５０μｇ·ｋｇ￣（－１）自兔耳静脉注入引起心肌ｃＡＭＰ浓度升高，肌膜Ｃａ￣（２＋）－ＡＴＰ酶及超氧化物歧化酶（ＳＯＤ）活性减低，预先ｉｖ硫酸镁可以有效地阻抗ＮＥ引起的这些心肌生化改变，表明镁对ＮＥ体内水平过高时引起的心肌膜酶损害具有一定的保护作用。
The intranuclear injection of 50μg · kg -1 norepinephrine (NE) caused the increase of myocardial cAMP concentration and the decrease of Ca 2+ -ATPase and SOD activity iv Magnesium sulfate effectively counteracts these myocardial biochemical changes caused by NE, suggesting that magnesium has a protective effect on myocardial membrane enzymes induced by excessive levels of NE in vivo.