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作者等用钥孔(虫戚)血兰蛋白使鼠致敏,然后用同样抗原贴附于其暴露的牙髓上,用病理组织学和免疫组织学方法检查牙髓的改变,发现:1.牙髓在贴附抗原24小时后,嗜中性白细胞浸润达到高峰,露髓下方的小血管周围可见IgG 抗原和纤维蛋白样退变,而未致敏鼠或贴附生理盐水的牙髓只有轻度炎症改变,提示阿尔图斯(arthus)反应可能是牙髓炎的形成机制;2.Ia 抗原原阳性细胞或巨噬细胞在24小时也有增加,实验期内未
The authors used the keyhole (Qi Qi) hemagglutinin sensitized mice, and then with the same antigen attached to the exposed pulp, using histopathological and immunohistological examination of dental pulp changes found: 1. Twenty-four hours after attachment of antigens, the neutrophil infiltration peaked. Dental pulp showed degeneration of IgG antigen and fibrinogen around the small blood vessels below the exposed pulp, whereas the non-sensitized rats or saline-only pulp had only light Degree of inflammatory changes, suggesting that Arthus reaction may be the formation of pulpitis mechanism; 2.Ia antigen-positive cells or macrophages also increased at 24 hours during the experimental period