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心肌坏死是由于冠脉闭塞引起的氧供需平衡严重失调所致。粥样化损害、血管痉挛以及血小板激活因子等复杂的相互作用,可促使冠脉血栓形成以致闭塞。正常时,心肌功能取决于氧化磷酸化作用,氧供需失调使心肌细胞正常功能和代谢所需要的内环境稳定机理遭破坏。现已证实,梗塞范围与病死率有良好的相关性。因此,缩小梗塞范围可降低病死率。实验证明,凡能减少需氧、增加供氧、增加缺血区生能底物的输送、抗自溶或异溶过程等措施,皆可挽救缺血心肌。β肾上腺素能阻滞剂
Myocardial necrosis is caused by a serious imbalance of oxygen supply and demand due to coronary occlusion. Complex interactions such as atherosclerotic lesions, vasospasm, and platelet activating factor promote the formation of coronary artery thrombosis. Normal, myocardial function depends on the role of oxidative phosphorylation, oxygen supply and demand disorders so that the normal function of myocardial cells and metabolism required by the stability of the internal environment was destroyed. It has been confirmed that infarct size and mortality have a good correlation. Therefore, reducing infarct size can reduce mortality. Experiments show that all can reduce aerobic, increase oxygen supply, increase the transport of ischemic area of the substrate, anti-autolytic or iso-soluble process and other measures, can save ischemic myocardium. Beta-adrenergic blockers