一般认为咪唑奎宁通过细胞内信号通路介导炎症反应是其临床应用的基础,但也有报道其独立于免疫反应之外的作用,如降低细胞生存能力及诱导凋亡等,然其详细机制未知。研究者通过对人膀胱癌T24细胞系及鼠膀胱癌MBT-2细胞系的实验发现经咪唑奎宁处理后,T24细胞及MBT-2细胞的c-Myc的 It is generally believed that imidazole quinine mediates inflammatory response through intracellular signaling pathways, which is the basis of its clinical application. However, it has also been reported that it acts independently of the immune response, such as reducing cell viability and inducing apoptosis. However, its detailed mechanism is unknown . Researchers through the human bladder cancer T24 cell line and mouse bladder cancer cell line MBT-2 experiments found that after imidazole quinine treatment, T24 cells and MBT-2 cells c-Myc