钾离子(K+)是维持细胞膜内外电位差的重要离子,低血钾时,心肌细胞膜两侧K+浓度差增大,可诱发浦氏纤维和心室肌细胞产生后除极电位及触发活动,导致多种室性心律失常[1],但引起扭转型室速(torsade de pointes,TdP)者并不多见,现结合2例经治患者进行分析讨论。1 对象和结果 按照Horowitz等[2]诊断标准,2例患者均符合TdP。其病因、诱因、临床表现和心电图特征如下: Potassium (K +) is an important ion that maintains the potential difference between the inside and outside of the cell membrane. When hypokalemia is applied, the K + concentration difference on both sides of the myocardial cell membrane increases, which can induce the depolarization potential and triggering activities of Pu’s fibers and ventricular myocytes, A variety of ventricular arrhythmias [1], but caused by torsades de pointes (TdP) are rare, is now combined with 2 cases of patients treated for analysis and discussion. 1 Subjects and results According to Horowitz et al [2] diagnostic criteria, 2 patients were in line with TdP. The etiology, causes, clinical manifestations and ECG characteristics are as follows: