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在培养的家兔主动脉平滑肌细胞上观察到，纤维蛋白能够剂量依赖性地刺激平滑肌细胞增生，刺激胶原合成，增加细胞内胆固醇堆积，提示纤维蛋白很可能参与了动脉粥样硬化的发生。
It was observed on cultured rabbit aortic smooth muscle cells that fibrin stimulated smooth muscle cell proliferation in a dose-dependent manner, stimulated collagen synthesis and increased intracellular cholesterol accumulation, suggesting that fibrin is likely involved in the development of atherosclerosis.