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目的 建立猫的贲门失弛缓症动物模型并探讨一氧化氮 (NO)在贲门失弛缓症中所起的作用。方法 胃镜下将BAC (benzyldimethyltetrad -ecylammoniumchloride)环周注射到猫 (12只 )下食管括约肌 (LES)内 ,对照组 (6只 )注射生理盐水 ,8周后观察进食和体重改变 ;造影检查食管内钡剂
Objective To establish an animal model of achalasia in cats and to explore the role of nitric oxide (NO) in achalasia. Methods BAC (benzyldimethyltetrad-ecylammonium chloride) was injected into the lower esophageal sphincter (LES) under the gastroscope. The control group (6 rats) was injected with normal saline. After 8 weeks, the changes of food intake and body weight were observed. Barium