生姜对空腹时胆囊大小及试餐后胆囊收缩的影响

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使用超声成像仪研究生姜对人体空腹胆囊大小和试餐引起的胆囊收缩运动的影响。结果表明,人口服5%的生姜煎液100ml对空腹胆囊大小无明显影响,但可明显减少试餐引起的胆囊收缩。P<0.01;与对照组第3次探查相比ΛP<0.05结果表明,生姜对空腹胆囊大小无明显影响,但可明显减小试餐后胆囊短径的缩小,即对试餐引起的胆囊收缩有抑制作用。两组空腹第一次测定的胆囊最大长径与最大短径均无显著差异(P>0.05)。3讨论本研究结果表明,口服5%的生姜煎液100ml可明显减小试餐后胆囊的收缩。生姜(以及干姜)属温里药,中医用于胃寒呕吐、腹痛、腹泻等症[4、5]。现代药理学研究证明[6],从生姜中分离出来的姜油酮及姜烯酮的混合物,有止吐的效果,姜油酮可使肠管松驰、蠕动减弱;生姜浸膏能抑制硫酸铜引起的狗的呕吐。本研究结果与上述生姜的中药药理理论及理代药理研究结果相一致,即具有抑制消化道(包括胆囊)平滑肌运动的作用,其作用机理是否与影响消化道内PGS的合成与释放有关,仍需进一步研究。参考文献 Effect of Ultrasound Imager Graduate Ginger on Gallbladder Shrinking Movement Caused by Fasting Gallbladder Size and Dietary Test in Human Subjects The results showed that oral administration of 5% ginger decoction 100ml has no significant effect on the size of fasting gallbladder, but it can significantly reduce the gallbladder contraction caused by the trial meal. P<0.01; Compared with the third exploratory study, the ΛP<0.05 results showed that ginger had no significant effect on the size of fasting gallbladder, but it could significantly reduce the shortening of the short axis of the gallbladder after the test meal. The gallbladder contraction caused by inhibition. There was no significant difference in the maximum length of the gallbladder between the two groups (P>0.05). 3 Discussion The results of this study showed that oral administration of 5% ginger decoction 100ml can significantly reduce the gallbladder contraction after the trial meal. Ginger (as well as ginger) is a warming medicine. Chinese medicine is used to treat cold vomiting, abdominal pain, and diarrhea [4, 5]. Modern pharmacological studies have shown [6] that the mixture of zingerone and zingerone isolated from ginger has an antiemetic effect, zingerone can relax the intestinal tract, and the peristalsis is weakened; ginger extract can inhibit copper sulfate The vomiting caused by the dog. The results of this study are consistent with the above-mentioned pharmacological theory of traditional Chinese medicine and pharmacological studies of traditional Chinese medicines. They have the effect of inhibiting the movement of the smooth muscles of the digestive tract (including gallbladder). Whether or not the mechanism is related to the synthesis and release of PGS in the digestive tract is still needed. further research. references
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