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作者发现人类肥厚性心肌病(HCM)所表现的左室动力过度与叙利亚仑鼠遗传性心肌病所显示的心肌细胞对钙离子的摄入和含量增加相类似,因而应用一种减少钙离子通过细胞膜的药物—异搏定的治疗,从而防止心肌代谢和解剖学上的异常。 病例选择与方法:作者对2.7例HCM患者(基础状态下或激发后主动脉下收缩压阶差在30mmHg以上)作了血液动力学研究。异搏定静脉输入采用
The authors found that left ventricular overdrive exhibited by human hypertrophic cardiomyopathy (HCM) is similar to that of cardiomyocytes showing uptake and increased levels of calcium ions in Syrian-Allen heritable cardiomyopathies, and thus a reduction in calcium ion passage Cell membrane drugs - verapamil treatment, thereby preventing myocardial metabolism and anatomical abnormalities. Case Selection and Methods: The authors performed a hemodynamic study of 2.7 patients with HCM (basal or post-aortic systolic pressure gradient above 30 mm Hg). Verapamil intravenous input