近日研究指出:实验性冠状动脉阻塞后,缺血区某些心肌细胞虽处于缺血及失去功能状态,但在早期仍是可逆的。这些细胞若能存活,则梗死的面积便会减少。同时,从电生理学来看,这些细胞异常不稳定,常导致某些致死性心律紊乱,如能及时挽救这些濒危的心肌细胞,将使急性心肌梗塞的预后大为改观。一、息性冠状动脉阻塞后,缺血心肌形态学的早期变化缺血心肌早期形态学的改变在人类急性心肌梗塞常无机会进行观察研究,有关的研究资料 Recent studies have pointed out: experimental coronary artery occlusion, ischemic area, although some of the myocardial cells in ischemia and loss of function, but still reversible in the early. If these cells survive, the infarct size will be reduced. At the same time, from the electrophysiological point of view, these cells abnormal instability, often leading to some lethal heart rhythm disorders, such as timely rescue of these endangered cardiomyocytes, acute myocardial infarction will greatly improve the prognosis. First, the occlusion of interest-induced coronary artery occlusion, ischemic myocardial morphological changes in the early morphological changes of ischemic myocardium in human acute myocardial infarction often have no opportunity to observe the study, the relevant research data