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眼部感染HSV-1后,在支配该区域的三叉神经节的感觉神经细胞中建立潜伏感染,宿主的免疫因素和病毒的某些功能参与了潜伏感染的建立,潜伏病毒基因组大部分呈游离状态,仅用一小部分整合到宿主DNA中,并发生不同程度的转录,当遇到适宜刺激时潜伏病毒基因组可被激活,返回原发感染部位引起复发。重组DNA技术和组织培养技术的应用为潜伏感染的研究开辟了新的领域。
After eye infection with HSV-1, latent infection is established in sensory neurons that control the trigeminal ganglia in this area. Host immune factors and some functions of the virus are involved in the establishment of latent infection. Most of the latent virus genome is in a free state , Only a small part of the integration into the host DNA, and the occurrence of varying degrees of transcription, when faced with appropriate stimulation of the latent virus genome can be activated, return to the primary site of infection caused by recurrence. The application of recombinant DNA technology and tissue culture technology has opened up new fields for the study of latent infection.