肺水肿是恶性疟的一种严重和常为致死性的并发症。可因体液过多(尤为肾衰时)所致,但也可在无高容量的临床改变时发生。作者报道一例并发肺水肿患者在其发病机理中血液动力学因素大于体液过多。男、19岁,昏迷,体温38。50,颈软,二侧视乳头水肿,并有苍白、黄疽和肝脾肿大。血红蛋白3. 9克%,自细胞7,100,血小板60,000/立方毫米;I凝血酶原指数50％,纤维蛋白元降解产物增加’血尿素182毫克％,血 Pulmonary edema is a serious and often fatal complication of falciparum malaria. May be due to body fluids too much (especially when renal failure), but can also occur in the absence of high-capacity clinical changes. The authors report that a patient with concurrent pulmonary edema had a hemodynamic factor greater than that of bodily fluids in its pathogenesis. Male, 19 years old, coma, body temperature 38.50, neck soft, bilateral papillae edema, and pale, jaundice and hepatosplenomegaly. Hemoglobin 3.9%, since cells 7,100, platelets 60,000 / mm3; I prothrombin index 50%, fibrin degradation products increased ’blood urea 182 mg%, blood