许多实验工作者指出,血小板在内毒素引起的肺动脉高压具有重要作用,其机理有人解释为血小板对肺毛管床的机械阻塞或为继发性血小板释放血管活性物质所引起。有人进一步指出,在伴有败血症和败血性休克的呼吸衰竭,其发展过程血小板可能是关键环节。本文试 Many experimenters point out that platelets play an important role in endotoxin-induced pulmonary hypertension, a mechanism that has been explained by the mechanical blockage of platelets on the pulmonary capillary bed or the release of vasoactive substances from secondary platelets. It was further pointed out that in the respiratory failure associated with sepsis and septic shock, the development of platelets may be the key link. This article test