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肾脏处理钙和磷酸盐的能力,反映了甲状旁腺素(PTH)的活力。作者对18例急性胰腺炎病人入院后头5天内钙和磷酸盐值进行了测定。6例有低血钙症(Ca<1.08),并似低尿钙、低磷酸盐血症和低肾小管磷酸盐的再吸收,但其 PTH 值变异很大。一例患者发生了低钙性抽搐,静脉补充钙后,血浆钙升高,肾脏的变化即见逆转,但 PTH 值变化不大。作者未能证实低镁血症或 PTH 的蛋白水解性降解是低血钙症的病因。急性胰腺炎时的低钙血症是由多种因素造成的,其中脂肪坏死的钙沉着是主要原因。在某些病例,
The ability of the kidneys to handle calcium and phosphate reflects the viability of parathyroid hormone (PTH). The author of 18 cases of acute pancreatitis patients admitted to hospital within the first 5 days of calcium and phosphate values were measured. Six patients had hypocalcemia (Ca <1.08) and were hypoglycaemic, hypophosphatemic and low renal tubular phosphate resorption, but their PTH values varied widely. A case of hypocalcemic convulsions, intravenous calcium supplementation, the plasma calcium increased, the change of the kidney is reversed, but little change in PTH. The authors failed to confirm that hypomagnesemia or proteolytic degradation of PTH is the cause of hypocalcaemia. Hypocalcemia in acute pancreatitis is caused by a variety of factors, of which calcification of fat necrosis is the major cause. In some cases,