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长期接触低浓度的镉可致肾功能障碍。蛋白尿、Fanconi氏症候群及比较少见的氮血症,被认为是镉肾症的临床表现。迄今用于检测早期镉源性肾功能障碍的实验方法均不够满意。 尿的细胞学检查已用于多种肾病的早期检测,尤其在铅性肾损害的检测上已初步引起注意。由于镉肾症和铅肾症在发病机理上有相似之处,作者将尿细胞学检查与目前沿用的其它检测方法进行了比较,以探讨其实用价值。 调查对象为21名从事钡/镉硬脂酸盐和镉硒颜色生产的男性工人,并在同一公司内以23名未接触过镉且无肾病史的男性工人作对照。
Long-term exposure to low concentrations of cadmium can cause renal dysfunction. Proteinuria, Fanconi’s syndrome and, more rarely, azotemia are considered as clinical manifestations of cadmium and kidney disease. So far, the experimental methods used to detect early cadmium-derived renal dysfunction are not satisfactory. Urine cytology has been used for the early detection of a variety of kidney diseases, especially in the detection of lead kidney damage has initially attracted attention. Because of the similarities in the pathogenesis of cadmium and kidney disease and lead-induced kidney disease, the authors compared urinary cytology to other currently available tests to explore its practical value. The survey consisted of 21 male workers in barium / cadmium stearate and cadmium selenium production and compared them to 23 male workers in the same company who had not been exposed to cadmium and had no history of kidney disease.