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急性心肌梗塞(AMI)时室性过早搏动(PVC)是常见的,通过急性电生理实验和对缺血心肌的代谢改变的观察,已表明两者间的相互关系。由于PVC可导致危险的心律紊乱,例如室颤。所以在AMI时出现PVC具有临床的重要意义。PVC也可见于慢性心肌缺血,例如陈旧性心肌梗塞,Wellens等提出AMI时室性心律紊乱的形成机制是自发的(automatic mechanisin);陈旧性心肌梗塞者则最可能是折返激动(reentry circuit)所致;PVC也可见于无品质性心脏病者。
Ventricular premature beats (PVCs) are common in acute myocardial infarction (AMI) and the correlation between the two has been shown by acute electrophysiological studies and observation of metabolic changes in ischemic myocardium. PVC can cause dangerous heart rhythms, such as ventricular fibrillation. Therefore, PVC appears clinically important in AMI. PVC is also seen in chronic myocardial ischemia, such as old myocardial infarction. The mechanism by which Wellens et al. Proposed ventricular arrhythmia when AMI was presented was automatic (mechano- mechaninin); those with old myocardial infarction were most likely reentry circuits Due to PVC can also be found in those who have no heart disease.