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疟疾并发贫血的机理尚未完全明了。急性期疟原虫破坏红细胞,但患者经治疗原虫血症消失后,溶血现象仍持续存在。可能免疫过程起重要作用。作者在法国一医院对曾住在加蓬的14例慢性疟疾患者进行了研究。其中女性11例,男性3例。年龄15～66岁。患者分别因贫血(2)、继发性不孕(2)、肾功能不全、截瘫、上肢修复术、耳聋、二尖瓣狭窄(2)、慢性骨关节病、心肌病、静脉炎及体格检查而入院。均无血液病和肿瘤。2例贫血患者有发热。6例脾肿大,4例肝肿大。入院前均未服过疟疾预防药。厚薄血膜检查疟原虫全部阴性,3例经Ficoll血浓集检查亦阴性。
The mechanism of anemia complicated by malaria is not fully understood. Acute malaria parasites destroy erythrocytes, but hemolysis persists even after treatment of parasitemia disappears. It may play an important role in the immune process. The authors studied 14 patients with chronic malaria who had lived in Gabon in a French hospital. There were 11 females and 3 males. Age 15 ~ 66 years old. Patients were diagnosed with anemia (2), secondary infertility (2), renal insufficiency, paraplegia, upper extremity repair, deafness, mitral stenosis (2), chronic osteoarthritis, cardiomyopathy, phlebitis and physical examination And admission. No blood diseases and tumors. 2 cases of anemia in patients with fever. 6 cases of splenomegaly, 4 cases of hepatomegaly. Have not taken malaria prophylaxis before admission. All blood samples were negative for Plasmodium, and 3 were negative for Ficoll concentration.