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目的:观察健康受试者在运动性肌肉疲劳过程中大脑中枢电信号与外周肌肉表面肌电电信号通讯之间的变化,为丰富运动性肌肉疲劳的中枢机制理论提供参考依据。方法:以15名健康受试者为研究对象,通过上肢间断性握力收缩诱发其指屈肌疲劳,观察疲劳过程中运动皮质区的β波段(15~30 Hz)脑电、指深屈肌肌电以及脑电与肌电相干性的变化。结果:在疲劳过程中位于FC1、FCz、FC2、C1电极位置的β波段标准化能量谱显著性下降(P<0.05);指深屈肌s EMG的15~30 Hz标准化能量谱随着疲劳发生显著下降(P<0.05)。FC1、FCz、FC2、C1以及Cz电极位置的β波段脑电-肌电相干性随着疲劳的加重显著性下降(P<0.05)。结论:在小强度运动诱发上肢肌肉疲劳的实验中,β波(15~30 Hz)频率带的EEG和EMG功率谱随着疲劳的加重而显著性降低,同时β波(15~30 Hz)频率带对应的EEG-EMG相干性显著性下降。
OBJECTIVE: To observe the changes of electrical signals between central brain electrical signals and EMG signals during exercise-induced muscle fatigue in healthy subjects, and provide a reference for enriching the central mechanism of exercise-induced muscle fatigue. Methods: Totally 15 healthy volunteers were enrolled in this study. Their finger flexor fatigue was induced by intermittent grip contraction of the upper extremity. The β-band (15-30 Hz) EEG in the motor cortex during fatigue was observed. Electrical and EEG and EMG related changes. Results: The normalized energy spectra of β-band at the positions of FC1, FCz, FC2 and C1 were significantly decreased during fatigue (P <0.05). The normalized energy spectrum of 15-30 Hz Decreased (P <0.05). The β-band EEG-relatedness of FC1, FCz, FC2, C1 and Cz electrodes decreased significantly with fatigue (P <0.05). CONCLUSION: The power spectrum of EEG and EMG in the frequency band of β wave (15-30 Hz) decreases significantly with fatigue, while the frequency of β wave (15-30 Hz) The corresponding EEG-EMG coherence decreased significantly.