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急性心肌梗塞时,支配心脏的交感神经和副交感神经功能失调,交感神经活动增强,引起窦性心动过速。副交感神经的过度活动可引起窦性心动过缓。二者都会给心肌梗塞患者带来严重后果。当某一支冠状动脉闭塞后,血流中断,该支动脉供血的心肌缺血、缺氧、损伤、坏死。发病早期,植物性神经功能零乱;引起窦性心动过速时,能使梗塞区周围心肌,供氧与耗氧之间的平衡进一步遭到破坏,引起梗塞区边缘心肌的坏死。其梗塞范围越大,发生窦性心动过速的可能性就越大。而窦性
Acute myocardial infarction, the heart of the sympathetic and parasympathetic dysfunction, increased sympathetic activity, causing sinus tachycardia. Parasympathetic over-activity can cause sinus bradycardia. Both will have serious consequences for patients with myocardial infarction. When a coronary artery occlusion, blood flow interruption, the arteries to provide myocardial ischemia, hypoxia, injury, necrosis. Early onset, vegetative nervous disorder; cause sinus tachycardia, can make the myocardial infarction area around the balance between oxygen and oxygen was further damaged, causing myocardial infarction in the edge of the area of necrosis. The greater the extent of its infarction, the greater the likelihood of sinus tachycardia. And sinus