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变异型心绞痛发作时表现为ST段抬高者,其冠状功脉有严重痉挛,有些病例则表现为ST段下降,这种动力性冠状动脉狭窄对引起心肌缺血起着重要作用。实验和临床结果提示,ST段的偏移可能不仅依赖于血管痉挛的严重程度和部位,而且有赖于侧枝循环发展的范围。但由于侧枝血管出现短暂,加以痉挛和非痉挛动脉提供侧枝血流的观察技术上的困难,常不易证实。本文报道三例劳力型及安静型心绞痛,当用麦角新碱诱发冠脉痉挛时,短暂观察到由非痉挛动脉向缺血区形成的侧枝血管。
Variant angina pectoris occurs when the ST-segment elevation, the corpus sac of severe spasm, while in some cases showed ST-segment decline, this dynamic coronary artery stenosis plays an important role in causing myocardial ischemia. Experimental and clinical findings suggest that the shift in ST segment may depend not only on the severity and location of vasospasm but also on the extent of collateral circulation development. However, because of the transient presence of collateral vessels, the technical difficulties of observing collateral blood flow with spastic and non-convulsive arteries are often difficult to verify. We report three cases of labor-type and silent angina pectoris. When ergometrine-induced coronary artery spasm was used, collateral vessels formed from the non-convulsive artery to the ischemic area were briefly observed.