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作者结合动脉粥样硬化的发病机制观察了Vit.E对大鼠实验性动脉粥样硬化的抑制作用。结果表明Vit.E在降低血清总胆固醇、血清过氧化脂质和升高动脉壁PGI_2的同时,对大鼠实验性动脉粥样硬化的病变的形成有明显的抑制作用。并且有拮抗钙的作用,能抑制钙沉着于动脉壁。
The authors observed the inhibitory effect of Vit. E on experimental atherosclerosis in rats in combination with the pathogenesis of atherosclerosis. The results showed that Vit.E had a significant inhibitory effect on the formation of experimental atherosclerotic lesions in rats while reducing serum total cholesterol, serum lipid peroxides, and increasing arterial wall PGI2. It also acts as an antagonist of calcium and inhibits calcium deposition in the arterial wall.