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两文分别对慢性中耳炎及胆脂瘤所造成的中耳骨质吸收进行了组织学研究,以探讨中耳炎病程中骨质吸收的机理。研究材料为慢性中耳炎听骨病变边缘区的粘膜下层-骨组织及胆脂瘤骨疡边缘区的粘膜下层-骨组织。前者系中耳术中取出之砧骨;后者为中耳术中或活检采取之骨质。经电子显微镜观察的结果是:病理砧骨的粘膜下层与坏死骨质间的边缘区有明显的毛细血管增生及细胞增生,主要是组织细胞增生,其间质有明显的胶原溶解现象。胆脂瘤骨疡边缘区的粘膜下层及骨组织所见与前者相同。作者们从而得出以下结论:慢性中耳炎的骨质吸收并非如以往学者们所公认的是由于血运不佳、组织缺氧造成;胆脂瘤形成的骨质吸收亦并非由于胆脂瘤对骨质的压迫吸收所
The two groups of chronic otitis media and cholesteatoma were caused by the middle ear bone absorption histological study to explore the course of otitis media bone absorption mechanism. The research material was the submucosa-bone tissue and the submucosa-bone tissue in the edge of the sore of the cholesteatoma in the marginal zone of otitis media with chronic otitis media. The former is the removal of the incus in the middle ear surgery; the latter is the middle ear surgery or biopsy to take the bone. The results of electron microscopy: pathological incarceration of the submucosa and necrotic border between the obvious capillary proliferation and cell proliferation, mainly the proliferation of tissue cells, the interstitial obvious collagen dissolution phenomenon. Cholesteatoma margins of bone mucosa and bone tissue seen in the same with the former. The authors thus conclude that the bone resorption of chronic otitis media is not as recognized by previous scholars as due to poor blood circulation and hypoxia; the bone resorption of cholesteatoma is not due to the effect of cholesteatoma on bone Quality oppression absorption