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我们曾观察到消炎痛预处理能使大鼠对半乳糖胺肝损伤产生明显的保护作用,本研究利用大鼠肝细胞原代培养的方法进一步观察了达一现象。结果提示,消炎痛整体处理可使肝细胞本身获得抗损伤的能力,而这种抗损伤能力的形成可能有赖于肝细胞外其它因素的参与。
We have observed that pretreatment with indomethacin can significantly protect rats from galactosamine-induced hepatic injury. In this study, the primary culture of rat hepatocytes was used to further observe the phenomenon. The results suggest that the overall treatment of indomethacin can gain the ability of liver cells to resist damage, and the formation of this anti-injury ability may depend on other factors outside the liver cells.