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锰作业锰中毒人员共33例，脑运动传导时间和常规肌电图检查，并与临床体征相匹配分析，结果表明：临床症状和体征随着锰作业时间延长而加重，主要为肌力增高，昂佰征阳性，工龄≥30年者和锰中毒者脑运动传导时间明显延长（P＜0．001），而1～29年者正常但较健侧有延长。工龄≥30年及锰中毒者常规肌电图时限延长，电压增高，则1～29年者时限缩短，电压低，所有病人多项电位增多，大部分重收缩定量分析为肌源性改变。结果说明：（1）从事作业不宜多于30年；（2）早期为锥体外系统损害，晚期才出现锥体束的损害；（3）锰作业人员测定脑运动传导时间可作为锰中毒诊断力法之一，亦可作为锰作业评定锥体束损害之依据．
Manganese poisoning of manganese workers in a total of 33 cases, brain conduction time and conventional electromyography examination, and clinical signs and match analysis, the results showed that: clinical symptoms and signs increased with the extension of manganese work time, mainly for increased muscle strength, AMBI positive, longer than 30 years of age and manganese poisoning significantly longer time to brain conduction (P <0.001), while 1 to 29 years were normal but longer than the healthy side. Long service life of more than 30 years and conventional electromyography manganese poisoning time limit, the voltage increases, then 1 to 29 years shorten the time, low voltage, all patients increased multi-potential, most of the shrinkage quantitative analysis of myogenic changes. The results showed that: (1) the operation should not be more than 30 years; (2) the early damage to the extrapyramidal system, the cone injury occurred late; (3) Manganese workers determine the brain conduction time can be used as manganese poisoning diagnostic power One of the methods can also be used as a basis for assessment of pyrite damage caused by manganese work.