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近来研究发现,部分支气管哮喘(简称哮喘)的不可逆性气流阻塞和持续性气道高反应性,与气道重塑有关[1]。虽然哮喘气道重塑发生机制复杂,可能与上皮间质的相互作用导致胶原增多这一中心环节有关。其中基质金属蛋白
Recent studies have found that some of the bronchial asthma (asthma) irreversible airflow obstruction and persistent airway hyperresponsiveness, and airway remodeling [1]. Although the mechanism of airway remodeling in asthma is complex, it may be related to the central part of the collagen that leads to the interaction of epithelial mesenchyme. One of the matrix metalloprotein