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目的本文主要对高压氧预适应对神经系统保护作用的研究及其机制作一个简要的总结。高压氧预适应可明显诱导脊髓、心肌、肝脏、肾脏、肺组织、骨组织等器官和组织对损伤的耐受,但对神经系统其保护机制的研究目前还较少,具体的分子机制尚不明确,可能是通过减少神经细胞的凋亡及减轻脑水肿诱导神经保护效应,目前仍处在初步推测阶段。深入研究高压氧预适应的神经保护作用及其具体分子机制,将为今后其在高风险手术中的应用提供前提基础,并极大提高大手术的安全性,同时也可为以后开发内源性神经保护药物提供实践依据。
Aims This article mainly summarizes the research on the neuroprotective effects of hyperbaric oxygen preconditioning and its mechanism. Hyperbaric oxygen preconditioning can obviously induce the injury of the spinal cord, myocardium, liver, kidney, lung, bone and other organs and tissues, but there are still few studies on its protective mechanism in the nervous system. The specific molecular mechanism is not yet Clearly, may be through the reduction of nerve cell apoptosis and reduce brain edema induced neuroprotective effect, is still in the initial stage of speculation. In-depth study of the neuroprotective effect of hyperbaric oxygen preconditioning and its specific molecular mechanism will provide the premise for its application in high-risk surgery in the future, and greatly improve the safety of major surgery, but also for the future development of endogenous Neuroprotective drugs provide a practical basis.