脑损伤与严重低血糖持续的时间有关,而与发病次数无关〔1〕其发生机制,尚未见阐述。现将作者检索到的有关损害机制做如下归纳:据国外研究结果〔2〕,在进行胰岛素治疗的病人出现低血糖昏迷且脑电静止后,脑的氧消耗只有很小程度的减少,葡萄糖利用率减少了80%以上,而ATP水平仍维持 Brain damage and the duration of severe hypoglycemia, but has nothing to do with the frequency of occurrence [1] its mechanism has not been described. Now the author of the damage mechanism retrieved as follows: According to the results of foreign studies 〔2〕, in patients with insulin treatment of hypoglycemic coma and electroencephalostasis, the brain oxygen consumption decreased only to a small extent, glucose utilization Rate decreased by more than 80%, while the ATP level is still maintained