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前言口腔链球菌已被认为是龋病的主要病原因素,特别是变形链球菌。免疫学的研究已经证明,抗变链的抗体可以①抑制变链的生长和产酸。②凝聚变链的细胞。③抑制变链葡糠基转移酶(GTF)的活性。④干扰菌体定居于牙面。因而,在龋病发病的三因素基础上,加入了人体的免疫调节因素,使其更为全面(图1)。在血清中和唾液中的对链球菌的抗体,均可能影响口腔链球菌致龋性。血清中的免疫球蛋白可以在牙龈受损伤时进入口腔,如牙齿萌出、拔牙和各种临床治疗,也可通过龈沟液进入口腔。同时,牙龈和口腔组织的损伤也可以是致龋性菌休抗原进入人
Oral buccal mucosa has been considered as the main causal factor in dental caries, especially Streptococcus mutans. Immunological studies have shown that anti-chain variable antibodies can inhibit the growth of variable chains and acid production. ② condensation of chain cells. ③ inhibition of chain-changing activities of furfural transferase (GTF). ④ interfering bacteria settled in the tooth surface. Thus, in the pathogenesis of dental caries on the basis of three factors, adding the body's immune regulatory factors, to make it more comprehensive (Figure 1). Antibodies to streptococci in both serum and saliva may affect oral cariogenic cariogenicity. Immunoglobulins in serum can enter the oral cavity when the gums are damaged, such as tooth eruptions, tooth extraction and various clinical treatments, and also through the gingival crevicular fluid into the mouth. At the same time, gingival and oral tissue damage can also be caused by cariogenic bacterium antigens into humans