近二十多年来,休克的微循环障碍学说得到了证实与发展,并在休克抢救的临床实践中初获成效。但是随着认识的丰富与深入,进一步的研究又发现,休克时出现的很多代谢障碍,特别是细胞代谢异常,难以完全用微循环障碍学说来解释。 Nearly two decades, shock microcirculation theory has been confirmed and developed, and in the clinical practice of shock and rescue initially achieved results. However, with the enrichment and deepening of understanding, further studies have found that many metabolic disorders, especially cell metabolism abnormalities that occur during shock can hardly be completely explained by microcirculation disability theory.