在给小鼠１次静脉注射氯化错（６０ｍｇ／ｋｇ体重）后５ｍｉｎ至４８ｈ内的９个间期应用电镜结合Ｘ射线微区分析术，对镨在肝细胞与枯否细胞中的运转进行了追踪观察。在５ｍｉｎ～２ｈ，两种细胞均以胞吞方式摄取肝血窦中含镨微粒，在胞质中形成吞噬体。在吞噬体内，微粒群处于由稀疏至密集的浓缩过程，小吞噬体互相融合。这种胞吞作用在枯否细胞极为活跃，在肝细胞则较微弱。在４～４８ｈ，许多枯否细胞的胞质几乎被吞噬体充满，细胞变性；肝细胞内的吞噬体汇聚于胆小管周围。在胆小管腔内可见高电子致密微粒群，表明镨可经胆汁途径排出。 Electron microscopy and X-ray microanalysis were performed on 9 intervals within 5 min to 48 h after 1 intravenous injection of chloride chloride (60 mg / kg body weight) for the operation of praseodymium in hepatocytes and Kupffer cells Follow up observation. In 5min ~ 2h, both cells were taken up by endocytosis hepatic sinusoid containing praseodymium particles, the formation of phagosomes in the cytoplasm. In the phagosome, the particle swarm is in a process of concentration from dense to dense, and the small phagosomes fuse with each other. This endocytosis is extremely active in Kupffer cells, but weaker in liver cells. At 4-48 h, the cytoplasm of many Kupffer cells were almost completely engulfed by phagosomes and the cells degenerated. The phagosomes within hepatocytes converged around the bile ducts. In the small tube cavity visible high electron dense particle group, indicating that praseodymium can be discharged through the bile route.