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TLR识别病原体相关分子模式,激活先天免疫,并诱导获得性免疫反应,在抵御外来病原体的过程中起重要作用。IFN-γ作为一强有力的巨噬细胞致敏因子,可有效地活化巨噬细胞和NK细胞,调节树突状细胞、T细胞及B细胞的功能,参与先天免疫和获得性免疫应答。在此过程中,IFN-γ通过Jak-stat信号途径诱导转录因子IRF-8(干扰素调节因子8)表达上调,而
TLR recognizes pathogen-associated molecular patterns, activates innate immunity and induces an adaptive immune response that plays an important role in defense against foreign pathogens. As a powerful macrophages sensitizing factor, IFN-γ can effectively activate macrophages and NK cells, regulate the function of dendritic cells, T cells and B cells, and participate in innate and adaptive immune responses. In the process, IFN-γ induces upregulation of the transcription factor IRF-8 (Interferon Regulatory Factor 8) through the Jak-stat signaling pathway, whereas