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新生儿缺氧血性脑病(HIE)不仅可引起围产期新生儿死亡,而且是新生儿期以后造成伤残儿童的主要原因之一。其发病机制复杂,随着现代免疫学和分子生物学的迅速发展,近几年大量研究证明,脑缺氧缺血后可激活一系列分子反应,一部分可进一步介导脑损伤,同时也有一部分对脑组织起着保护性作用。相关生物学研究,对HIE预后以及干预治疗均有十分重要意义。复习有关文献综述如下。
Neonatal hypoxic-ischemic encephalopathy (HIE) can not only cause perinatal neonatal death, but also one of the major causes of disabled children during the neonatal period. Its pathogenesis is complex. With the rapid development of modern immunology and molecular biology, a large number of researches in recent years have proved that a series of molecular reactions can be activated after hypoxic-ischemic brain injury and some can further induce brain injury, Brain tissue plays a protective role. Related biological studies, the prognosis of HIE and intervention are of great significance. Review the literature review is as follows.