论文部分内容阅读
Multiple sclerosis(MS)is an autoimmune demyelination disease.IL-17–producing Th17 cells play a critical role in the development of MS.
MicroRNA-30a alleviates autoimmune inflammation by reducing inflammatory T cell development
【摘 要】
：
Multiple sclerosis(MS)is an autoimmune demyelination disease.IL-17–producing Th17 cells play a critical role in the development of MS.
【机 构】
：
Institute of Neuroscience and Key Laboratory of Molecular Neurobiology of Minister of Education,Depa
【出 处】
：
The 6th FAONS Congress and 11th Biennial Conference of CNS(第
【发表日期】
：
2015年3期
其他文献
Activity-dependent synaptic plasticity plays a key role in the refinement of neural circuits and expression of behaviors during development.
Objective Numb is first identified as a cell fate determinant in drosophila and is essential for maintaining neural stem cells during neurogenesis and promoting neuronal development.
Objective Depression is a pervasive mental disorder and a significant contributor to the global burden of disease,characterized by enduring sadness,loss of interest or pleasure,decreased energy or fee
Central histaminergic and orexinergic systems both originate in the hypothalamus restrictedly but innervate almost the whole brain,including various subcortical motor structures.
Objective Multiple sclerosis(MS)is an inflammatory and neurodegenerative disease of the central nervous system that causes substantial morbidity.
Objective T-helper(Th)17 cells are a new member in CD4+T lymphocytes and have a strong proinflammatory property.
Objective We previously found that sustained expression of interleukin(IL)-1b in mouse brain led to reduction in plaque pathology associated with increased evidence of glial activation,a finding that
Accumulated evidence suggested that the disturbance of iron metabolism might be one of initial causes leading to the degeneration of dopaminergic neurons in the substantia nigra(SN)of Parkinsons disea
Frataxin(FXN)is a mitochondrial protein that is involved in mitochondrial iron homeostasis and iron-sulfur cluster biogenesis.Defective expression of FXN causes an autosomal recessive mitochondrial di