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为了探讨麻痹性贝毒在紫贻贝(Mytilus edulis)体内的降解情况,于2009年4月中旬在大连市大窑湾海域采集640只紫贻贝在实验室条件下进行人工解毒试验。结果表明:实验第0 d时紫贻贝闭壳肌中的麻痹性贝毒以neo-STX为主,占总毒素含量的96.59%;性腺中只检测到GTX1一种组分,含量为43.17μgSTXeq/100 g;而紫贻贝的内脏团中的麻痹性贝毒含量最高,为162.68μgSTXeq/100 g,组成成分也复杂,共检出7种组分;外套膜和鳃中未检测到任何形式的麻痹性贝毒组分。在降解试验过程中,紫贻贝闭壳肌内的麻痹性贝毒的含量变化较小,但出现GTX3新组分;内脏团中的麻痹性贝毒变化较复杂,降解速度较快,实验第30 d时,仅检出一种组分neo-STX,含量为48.37μgSTXeq/100 g,共降解70.98%;而性腺中的GTX1在实验第3 d以后再未检出。
In order to investigate the degradation of paralytic shellfish poisoning in Mytilus edulis, artificial juvenile detoxification was conducted in laboratory conditions in the middle of April 2009 in 640 sea mussels of Dayao Bay, Dalian. The results showed that neotetra-neoxynucleotidase (STX) -based neps was the most common paralytic shellfish poisoning agent on 96th day, accounting for 96.59% of the total toxins. Only one component of GTX1 was detected in the gonad, with a content of 43.17μgSTXeq / 100 g, while the mussel shellfish had the highest content of paralytic shellfish poisoning (162.68 μgSTXeq / 100 g), with complex composition and 7 components. No any form was detected in the mantle and gills Paralytic shellfish poisoning component. During the degradation test, the content of paralytic shellfish poisoning changed little in the mussel shell muscle, but the new component of GTX3 appeared. The change of paralytic shellfish poisoning in the visceral mass was complicated and the degradation rate was fast. In the experiment At 30 days, only one component of neo-STX was detected, with a content of 48.37μgSTXeq / 100g and a total degradation of 70.98%. GTX1 in gonads was not detected after the 3rd day of experiment.