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近年来幽门螺旋菌(Helocobacter pylori 简称 HP)与慢性胃炎和消化性溃疡的相关性已得到证实,但 HP 的确切作用尚存在争论。究竟 HP 是病因还是继发侵入者?换言之,是先有 HP 引起胃粘膜改变还是已破坏了胃粘膜易于 HP 的寄存和定植?人们早已注意到肝硬化患者由于肝功能受损、门脉高压等因素,使得胃粘膜和内环境发生继发性改变,致使胃炎、溃疡病的发生率增高。在这已经存在胃粘膜损伤的肝硬化患者,其 HP 感染率的高低将有助于我们从侧面探讨 HP 的真正作用。本文通过调查肝硬化患者 HP 感染情况,旨在从理论上
In recent years, the correlation between Helicobacter pylori (HP) and chronic gastritis and peptic ulcer has been confirmed, but the precise role of HP remains controversial. Whether HP is the cause or secondary intruder? In other words, is the first to cause gastric mucosal changes caused by HP or damaged gastric mucosa is easy to HP storage and colonization? It has long been noted in patients with liver cirrhosis due to impaired liver function, portal hypertension Factors that make the secondary changes in gastric mucosa and the environment, resulting in increased incidence of gastritis, ulcer disease. In patients with cirrhosis who already have gastric mucosal injury, the HP infection rate will help us to explore the true role of HP from the side. This article investigates the HP infection in patients with cirrhosis and aims to be theoretically