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本文对随机抽取的１２例上消化道疾病患者的胃粘膜的超微结构进行了观察，结果表明胃粘膜上皮细胞超微结构的改变与炎症程度有关，而与幽门螺旋菌（ＨＰ）的感染及数量无关。未发现ＨＰ直接侵蚀、破坏胃粘膜上皮细胞的直接证据，但发现少数区域胃粘膜上皮细胞的游离缘出现与ＨＰ表面密切接触的台基状结构，这一现象是否是ＨＰ对粘膜细胞的直接损害作用，目前尚不清楚。因此，我们认为胃粘膜表面微环境的改变是ＨＰ继发感染的条件而非结果。此外在胃粘膜上皮细胞内发现一种不明种类的链杆菌存在，并分别对该杆菌和ＨＰ的形态进行了观察。
In this paper, 12 cases of upper gastrointestinal diseases in patients with gastric ulcer ultrastructure was observed, the results show that gastric mucosal epithelial ultrastructure changes and the degree of inflammation, and Helicobacter pylori (HP) infection and Number has nothing to do. No direct evidence of HP erosion or destruction of gastric mucosal epithelium was found. However, it was found that the free margin of gastric epithelial cells in a few areas appeared to have a basement-like structure in close contact with the HP surface. Is this a direct injury to mucosal cells by HP? Role, it is not clear. Therefore, we believe that the change of the microenvironment of gastric mucosa surface is the condition of HP secondary infection rather than the result. In addition, an unknown species of Streptococcus was found in gastric mucosal epithelial cells, and the morphology of the bacillus and HP were observed.