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例1 女,28岁,社员,因服敌百虫中毒于1976年10月15日急诊入院。入院时病员已深度昏迷,呈严重肺水肿及休克状态。经按有机磷农药中毒抢救及抗休克、纠正肺水肿等措施,3小时后神志清醒,中毒症状基本消失,住院14天痊愈出院。1周后感双手指及腕关节无力,疼痛,动作不灵,继而下肢亦感无力,踝关节疼痛,运动障碍。病变逐渐延及肘、膝关节,肌力减退(Ⅳ度)。数天后出现四肢肌肉萎缩,上肢以手背蚓状肌和骨间肌及手掌大小鱼际肌萎缩明显,手掌变平;下肢腓肠朋萎缩,足下垂,行动困难,腱反射消失;皮肤发凉,粗糙,无汗。呈典型末梢神经炎变。给维生素B_1、维生素B_(12)、加兰他敏等治疗,并配合中医辨证施
Example 1 Female, 28 years old, members, due to trichlorfon trichlorfon poisoning in October 15, 1976 emergency admission. The patient has been deeply coma on admission, showing severe pulmonary edema and shock status. After pressing organophosphorus pesticide poisoning and anti-shock, to correct pulmonary edema and other measures, 3 hours after the conscious, poisoning symptoms disappeared, hospitalized 14 days cured. A week later, his fingers and wrist weakness, pain, movement is not working, followed by weakness in the lower limbs, ankle pain, dyskinesia. Lesions gradually extended elbow, knee, muscle weakness (Ⅳ degrees). A few days later appeared muscle atrophy of the limbs, the upper limb to the hind legs earthworm muscle and interosseous muscle and palmar muscle atrophy at the time of significant muscle palms flattened; lower limb sural collapse, foot drooping, difficulty in operation, tendon reflex disappears; Rough, no sweat. A typical peripheral neuritis change. To vitamin B_1, vitamin B_ (12), galantamine and other treatment, and with TCM syndrome differentiation