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内皮素（ＥＴ）刺激儿茶酚胺释放，并增强血管平滑肌对儿茶酚胺的反应性，可引起肾脏血管强烈收缩，减少肾血流量。机体微循环障碍及血栓素、转化生长因子β等损伤性物质增加均可刺激血管内皮细胞产生和释放ＥＴ。降钙素基因相关肽（ＣＧＲＰ）的作用与之正相反。我们通过观察
Endothelin (ET) stimulates the release of catecholamines and enhances the responsiveness of vascular smooth muscle to catecholamines, which can cause strong contraction of the renal blood vessels and reduce the renal blood flow. Body microcirculation disorders and thromboxanes, transforming growth factor β and other substances can increase the damage of vascular endothelial cells to produce and release ET. Calcitonin gene-related peptide (CGRP) has the opposite effect. We pass the observation