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本文对高海拔地区（２２６ｌｍ）急性呼吸衰竭患儿３７例进行了甲襞微循环多项指标观察，在２４至４８小时治疗前后与正常组对比。以微循环积分值表示微循环障碍程度、积分值越高微循环障碍越严重。结果表明：甲襞微循环血管形成、流速流态、周围状态等多项指标有明显改变、微血管动脉管径（输入支）明显收缩变细，微血管静脉管径（输出支）随缺氧程度加重逐渐扩张变宽，是对低氧血症环境的一种保护性应激反应、而微血管内血流速度减慢、血流摆动、停滞、红细胞重度聚集、袢周出血、渗出等改变使微循环正常流态发生紊乱、则是构成小儿急性呼吸衰竭微循环障碍的病理基础。
In this paper, 37 cases of children with acute respiratory failure in high altitude (226lm) were observed a number of indicators of the microcirculation of formazan, 24 to 48 hours before and after treatment compared with the normal group. Microcirculation integral value to indicate the degree of microcirculation, the higher the integral value of microcirculation more serious. The results showed that there were significant changes in many indexes, such as microcirculation angiogenesis, flow velocity and the surrounding state, the diameter of arteriole (input branch) conspicuously constricted and constricted, and the diameter of microvascular vein (output branch) increased with the degree of hypoxia Gradually widening and widening, is a protective stress response to hypoxemia environment, while the capillary blood flow velocity slows down, blood flow, stagnation, severe accumulation of red blood cells, peri-Zhou bleeding, exudation and other changes so that micro Circulating normal flow disorders, constitute the pathological basis for microcirculatory disturbances in children with acute respiratory failure.