论文部分内容阅读
第一部分甲状腺与甲状旁腺疾患一、甲状腺妊娠期基础代谢率增加20～25％。此时甲状腺因滤泡上皮增生,滤泡体积与数目增加和血供增多而增大。因妊娠甲状腺肿常发生于低碘摄入地区,相对的碘缺乏可能是一种刺激。妊娠期碘代谢的改变首先由于肾小球滤过率增加(肾丢失碘增多),其次因母体碘池优先满足胎儿需要(经胎盘主动运转机制调节),可能引起母体碘缺乏。妊娠早期甲状腺素结合球蛋白(T_4BG)浓度增加2倍。通常血清总的 T_4、T_3和反 T_3升高。关于游离甲状腺激素浓度(FTHC)还未
The first part of the thyroid and parathyroid disorders First, the basal metabolic rate of thyroid pregnancy increased by 20 to 25%. Thyroid at this time due to follicular epithelial hyperplasia, follicular volume and the number increased and increased blood supply increased. Due to gestational goiter often occurs in areas with low iodine intake, the relative iodine deficiency may be an incentive. Changes in iodine metabolism during pregnancy first due to increased glomerular filtration rate (kidney loss of iodine increased), followed by the mother of iodine pool priority to meet the needs of the fetus (via the placental activation mechanism to regulate), may cause maternal iodine deficiency. Thyroxine binding globulin (T_4BG) concentration in early pregnancy increased by 2 times. Usually serum total T_4, T_3 and anti-T_3 increased. About free thyroid hormone concentration (FTHC) yet