论文部分内容阅读
胃幽门螺杆菌（HP）感染与消化性溃疡、胃淋巴瘤和胃癌等疾病的发生有关。该菌与胃上皮细胞作用的发病机制尚不十分清楚。尽管HP也被肯定是一种致癌因素，但其致癌机制据推测是HP诱导胃抗氧化作用减低，刺激上皮增生，从而造成胃癌多阶段模型上的突变。粘膜上皮增生可能是对HP?
Helicobacter pylori (HP) infection is associated with the development of diseases such as peptic ulcer, gastric lymphoma and gastric cancer. The pathogenesis of the bacteria and gastric epithelial cells is not yet clear. Although HP has also been identified as a carcinogenic factor, its carcinogenic mechanism has been postulated to be due to a decrease in HP-induced gastric antioxidant activity stimulated by epithelial hyperplasia resulting in a multi-stage model of gastric cancer. Mucosal epithelial hyperplasia may be on HP?