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急性心肌梗死(AMI)是当前人类医学界面临的严峻问题,从分子角度来说,其病因并不十分明确,并且病损机制复杂,从字面意义上来说,使缺血心肌尽快恢复血流(再灌注)是减轻AMI的理想措施。但人们发现,再灌注心肌时会引起严重的心肌死亡,即心肌再灌注损伤[1],随着医学的深入发展,人们发现再灌注损伤不仅仅局限于心肌,而是对全身各脏器均有影响,但其发病机制目前尚不十分清楚,可能
Acute myocardial infarction (AMI) is a serious problem currently faced by the human medical community. From a molecular point of view, the etiology of acute myocardial infarction (AMI) is not very clear, and the mechanism of the lesion is complex. In a literal sense, the ischemic myocardium resumes blood flow as soon as possible Reperfusion) is to reduce the ideal measure of AMI. However, it has been found that myocardial reperfusion can cause severe myocardial death, which is myocardial reperfusion injury [1]. With the further development of medicine, it has been found that reperfusion injury is not limited to myocardium, Affect, but its pathogenesis is not yet very clear, possible