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在23只麻醉猫,三碘季铵酚制动下,观察左心房和膀胱内感受器受刺激时,肾交感神经传出放电的变化。发现用50～80g重锤牵拉左心房1min期间,肾交感神经传出放电频率较对照值减少9.0～15.2％(p<0.01),切断两侧颈部迷走神经后,此效应明显减弱。用38℃生理盐水充胀膀胱10S,肾交感神经传出放电较对照值增加50.5％(p<0.01),切断支配膀胱的神经后,反应基本消失,对上述现象产生的机制和功能意义进行了简要的讨论。
Under the anesthesia of 23 cats and triiodide quaternary ammonium phenol, the change of the renal sympathetic nerve was observed when the left atrium and the receptors in the bladder were stimulated. It was found that the frequency of renal sympathetic outflow was reduced by 9.0-15.2% (p <0.01) during the 1 min pull-out of the left atrium with a weight of 50-80 g. This effect was significantly attenuated after the bilateral vagus nerves were severed. With 38 ℃ physiological saline inflated the bladder 10S, the renal sympathetic nerve discharge increased by 50.5% (p <0.01) compared with the control value. After cutting off the nerves that control the bladder, the reaction basically disappeared. The mechanism and functional significance of the above phenomenon Brief discussion.