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2000多年前,亚里斯多德认为,痛觉与触觉是同一事物的两个极端,适量的刺激是一种快意的接触,过多时则产生痛感.今天我们知道,认知痛楚并非如此轻而易举,当我们体验痛楚的时候,起作用的并不仅仅是受损组织所发出的大量神经信号.组织受损后激发整个专司此职的神经网络,传载神经信号在体内(脑中)行进,其传导路径与其他感觉信号有所不同.这些区别在神经系统最远端即已出现,锤子敲击的力量、极冷极热以及由受损细胞所释放的化学物质可将C神经纤维及A神经纤维激活,另一神经纤维A—β纤维传载非痛觉信号,这3种纤维汇集于脊髓,由神经元将传递来的信息自该处传往大脑.
More than 2,000 years ago, Aristotle thought that pain and touch are the two extremes of the same thing. The right amount of stimulation is a pleasure touch and the pain when it is too much. Today we know that cognitive pain is not so easy when we When you experience a pain, it is not just the large number of nerve signals that a damaged tissue is working on. The neural network that motivates your entire specialist in tissue damage, carries nerve signals in the body (in the brain) and conducts Pathways are different from other sensory signals, these differences occurring at the farthest end of the nervous system, the power of hammer strikes, extremely cold and very hot, and chemicals released by damaged cells that bind C nerve fibers and A nerve fibers Activation, another nerve fibers A-β fibers carrying non-pain signals, these three fibers in the spinal cord, the information transmitted by neurons from the Department to the brain.