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[例1]男,42岁。因恶心、手抖、气促1个月于1981年1月21日入院。体检:贫血貌,血压140/80,无水肿。实验室检验:尿蛋白++,尿盘状电泳低分子蛋白。BUN 69mg/dl,Cr 8.3mg/dl,C_(cr)40L/24h。从4月29日起患者每日尿量虽>1,600ml,但4个多月内血钾持续>5.6～6.7mEq/L。给低钾饮食,每日静注速尿200mg,服降钾树脂和甘露醇,血钾未降至正常。血氯110mEq/dl,CO_2CP29Vol％±。血浆肾素基础状态0.3～0.34(正常0.55±0.47ng/ml/h),激发状态0.47～0.52(正常3.84±2.74)。血管紧张素Ⅱ基础状态0～3(正常4～4Bpg/ml),激发状态0～12(正常10～90)。尿醛固酮
[Example 1] Male, 42 years old. Due to nausea, hand trembling, shortness of breath 1 month on January 21, 1981 admission. Physical examination: anemia appearance, blood pressure 140/80, no edema. Laboratory tests: urinary protein ++, urinary disc electrophoresis low molecular weight protein. BUN 69 mg / dl, Cr 8.3 mg / dl, C_ (cr) 40 L / 24h. From April 29 since the daily urine output of> 1,600ml, but more than 4 months of continuous potassium> 5.6 ~ 6.7mEq / L. To a low-potassium diet, daily intravenous furosemide 200mg, serving potassium-lowering resin and mannitol, serum potassium did not fall to normal. Blood chlorine 110mEq / dl, CO_2CP29Vol% ±. Plasma renin basal state 0.3 ~ 0.34 (normal 0.55 ± 0.47ng / ml / h), the excitation state 0.47 ~ 0.52 (normal 3.84 ± 2.74). Angiotensin Ⅱ basic state 0 ~ 3 (normal 4 ~ 4Bpg / ml), the excitation state 0 ~ 12 (normal 10 ~ 90). Urinary aldosterone