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对大鼠离体灌注的脾脏标本,NPY除能直接引起剂量依赖性的收缩外,还能增强去甲肾上腺素的收缩作用和抑制异丙基肾上腺素的舒张作用。NPY对脾脏组织cAMP的生成无明显影响。
In spleen specimens isolated from rats, NPY can not only directly cause a dose-dependent contraction, but also can enhance the contraction of norepinephrine and inhibit the relaxation of isoproterenol. NPY had no significant effect on cAMP production in spleen.