本研究对在急性情况下10只由药理学诱发心脏衰竭的狗身上李代桃僵昢了由骨骼肌驱动的辅助心室(SMV)增加心脏输出量的能力。在每只狗身上,通过在插入左心室尖动脉血管导管的可压缩囊周围覆盖未经训练的腹直肌,以外科手术方法建成了SMV。然后,在心室舒张期,多个运动元神经随着自然心室 This study demonstrates the ability of Lee to breach skeletal-muscle-facilitated ventricular (SMV) cardiac output in 10 acutely pharmacologically induced dogs with heart failure. On each dog, SMVs were surgically created by covering the untrained rectus abdominis around a compressible balloon inserted into the left ventricular apex vascular catheter. Then, during diastole, multiple locomotion neurons follow the natural ventricles