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阻塞性睡眠呼吸暂停低通气综合征(OSAHS)患者由于反复呼吸暂停导致缺氧/复氧,增加的氧化应激,致使一些氧化还原敏感的转录因子可能被激活,并通过增加其下游基因产物的转录而在OSAHS的病理过程中发挥重要作用。
Some patients with obstructive sleep apnea-hypopnea syndrome (OSAHS) due to repeated apnea lead to hypoxia / reoxygenation, increased oxidative stress, resulting in the activation of some redox-sensitive transcription factors and by increasing their downstream gene products Transcription and play an important role in the pathological process of OSAHS.