结果提示:急性运动过程中线粒体离子代谢紊乱抑制线粒体氧化磷酸化过程,减少 ATP 生成,进而造成线粒体肿胀、嵴破坏,进一步抑制氧化磷酸化,加剧离子代谢紊乱的“恶性循环”可能是运动性骨骼肌疲劳的重要原因。耐力训练通过提高线粒体的代谢机能,改善运动中线粒体离子代谢,以维持其正常形态可能是耐力训练增强机体抗疲劳能力,提高运动机能的重要机制。 The results suggest that the disturbance of mitochondrial oxidative phosphorylation during mitochondrial oxidative phosphorylation and the reduction of ATP production in mitochondria may lead to swelling and crest destruction of mitochondria, further inhibition of oxidative phosphorylation and aggravating the disturbance of ion metabolism in the process of acute exercise. An important reason for muscle fatigue. Endurance training can improve mitochondrial ion metabolism during exercise to maintain its normal morphology by improving mitochondrial metabolism and may be an important mechanism of endurance training to enhance the body’s anti-fatigue ability and improve motor function.